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Frataxin-knockout mouse myoblasts, heart, and skin fibroblasts all showed characteristics of ferroptosis, and Nrf2 was downregulated in Friedreichs Ataxia patients and frataxin-knockout mice, while Nrf2 activators EPI-743 or sulforaphane regulated redox imbalances and rescued ferroptosis in frataxin knockout cells

Proc Biochem 45:441445

This means that during times of high physical stress, heavy exercise, or recovery, your body may require more than it can naturally produce

These require close medical supervision due to their systemic effects and potential risks

Once diagnosed, treatment options include antiviral medications that can help reduce the severity and duration of outbreaks