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Thus, platelet activation may be associated with several factors, including abnormal RBCs, RBC exposed phophotidylserine, micropartcles, hemolysis, iron overload and ROS resulting in chronic platelet activation and hypercoagulability in -thalassemia/HbE patients (Fig

The treatment of secondary hemochromatosis

SLC1A1 ) or glutamate/aspartate transporter (GLAST