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Concurrent disruption of the AMPKNrf2 axis further weakens antioxidant capacity and exacerbates mitochondrial dysfunction

In the peroxisome proliferator-activated receptor (PPAR)-induced COPD model, upregulation of GPX3 expression can reduce lung tissue damage caused by oxidative stress (Reddy et al., 2018)

Two neuronal subpopulations present modifications in activity due to signals from the periphery, which includes the pancreas, adipose tissue, and the gastrointestinal tract ( Figure 1 One of these populations is represented by appetite-suppressing neurons, which function in these ways: 1

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Psychiatry 175 (7), 631648