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In ARDS, an imbalance between these pathways contributes to exaggerated immune responses, tissue damage, and disease progression ( 2.1 Ferroptosis: a central player in ARDS pathogenesis Ferroptosis is a regulated, iron-dependent form of cell death distinguished by oxidative damage and lipid peroxidation

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The faster onset with IV reflects near-complete bioavailability versus the 2535% absorption rate of even high-quality oral formulations

Integrated approaches, such as transcriptomic and bioinformatics analyses, have identified key immune cell-related genes (CXCL12, PDGFRL, AGTR1, PTGER3, S1PR1) with robust diagnostic potential, and emerging strategies like miRNA signatures and immunophenotyping of menstrual effluent may enhance diagnostic accuracy