[DOI] [PMC free article] [PubMed] [Google Scholar] 37.Yirmiya R., Goshen I
Poster Session II - DIGITAL TRANSFORMATION, AI AND ROBOTICS 07.00 - DIGITAL TRANSFORMATION, AI AND ROBOTICS - 07.01 - TECHNOLOGY INNOVATIONS: ROBOTS, VIRTUAL REALITY, ARTIFICIAL INTELLIGENCE AND MORE P911 - ESOC25-2156 NOVEL AI-POWERED, TIME-ADJUSTED NET WATER UPTAKE IMAGING BIOMARKER DERIVED FROM NON-CONTRAST COMPUTED TOMOGRAPHY TO VISUALIZE EARLY INFARCTED TISSUE AND PREDICT REVERSAL POTENTIAL Jonathon Cummock 1,2,3 , Timea Hodics 4 , Rahul Ghosh 1,2,3,5 , John Volpi 4 , Stephen Wong 2,3,6 , Kelvin Wong 2,3,6 1 Texas A&M University College of Medicine, Bryan, United States, 2 Houston Methodist Hospital, Department of Systems Medicine and Bioengineering, Houston, United States, 3 Houston Methodist Academic Institute, The Ting Tsung and Wei Fong Chao Center for BRAIN, Houston, United States, 4 Houston Methodist Hospital, Department of Neurological Surgery and Neurological Institute, Houston, United States, 5 Texas A&M University College of Engineering, Department of Biomedical Engineering, College Station, United States, 6 Houston Methodist Hospital, Department of Radiology, Houston, United States Background and Aims: In acute ischemic stroke (AIS), net water uptake (NWU) is a method to quantify tissue edema to detect and visualize ischemia
To enhance the therapeutic efficacy of neutrophil-based drug delivery systems (DDSs), researchers have suggested the in situ hitch-hiking of circulating neutrophils for cancer therapy
The copper-peptide complex stimulates collagen and glycosaminoglycan synthesis, promotes angiogenesis, activates antioxidant enzymes including superoxide dismutase, modulates inflammatory responses, and stimulates hair growth through follicle activation
Second, we provide a systematic and updated overview of glial contributions, highlighting emerging evidence that microglia act as primary drivers of iron-mediated neurotoxicity, and that astrocytes undergo dynamic phenotypic shifts regulated by pathways such as mTOR and CSE/YAP/FOXD3 mechanisms that modulate neuronal vulnerability in previously underappreciated ways
