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Virus infection was reported to reduce 7-dehydrocholesterol reductase (DHCR7) expression, the resultant higher levels of 7-DHC enhanced type I interferon production and promoted virus clearance in macrophages 13

The high levels of H 2 S in periondontal pockets, hemoxidation and hemolysis activity against red blood cells and ability to induce apoptotic cell death are thought to contribute to periodontal pathology [[55], [56], [57]]

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