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Hong Hy, Lee HY, Kwak W, Yoo J, Na MH, So IS, et al

The secretome of EAT mirrors that of dysfunctional VAT characterized by tumor necrosis factor-alpha, interleukin-6, and their respective soluble receptors [123] So, EAT promoted the development and maintenance of AF through a pro-inflammatory and pro-fibrotic environment [124] leading to autonomic imbalance, connexin dysfunction, cell shortening, increase collagen deposition, and calcium overload [125, 126]

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Funding This research was supported by the project of China Postdoctoral Science Foundation (No.2024M761881)

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