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4, administration of C10:2 markedly upregulated PCNA, cyclinA, CDK2, Bcl-2, MMP2, MMP9, TGF-, while significantly dowregulated Caspase3 and Bax in PASMCs compared with PDGF-BB group, indicating that C10:2 may promote PASMCs proliferation, apoptosis inhibition and extracellular matrix remodeling in vitro

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Similar to our observations regarding invasive infections, NKG2D KO mice were also significantly more susceptible to vulvovaginal Candida infection as seen by their increased fungal burden (Fig

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