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This biochemical mechanism supports the observed neuroprotective effects, as NAC enhances intracellular glutathione production, thereby reinforcing the cellular redox system, promoting detoxification of HNE, and ultimately protecting neurons from oxidative damage

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Laboratory studies demonstrate that combinations of improved senolytic peptides (such as ES2, which shows 3-7 times greater efficacy than FOXO4-DRI) with chemotherapeutic agents like Dabrafenib result in greater elimination of both cancer and senescent cells