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Associations between mitochondrial genetic variation, activation of the immune system, and its effects on neurogenesis and neurotransmission have led some authors to suggest that mitochondrial dysfunction initiates a chain of molecular events leading to key symptoms of neuropsychiatric long COVID, such as depressive mood and cognitive impairment [39, 100, 135]

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436 Recent work reveals that some Zur proteins harbor a pair of regulatory Zn II sites distinct from the central HHxHx 2 Cx 2 C motif ( vide supra ) that stabilizes an oligomeric assembly of three or more Zur dimers on the promoters of genes encoding cognate metal efflux transporters, leading to RNA polymerase recruitment and activation of transcription when metal levels become very high

In vivo studies using animal models or human volunteers will be employed to investigate its effects on gastric emptying, intestinal transit, and colonic motility