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Unlike sepsis, where treatments are primarily directed against LPS-induced inflammatory responses (e.g., anti-endotoxin antibodies), SAP-induced glycocalyx damage is predominantly driven by pancreatic enzyme activation (e.g., trypsin) and intestinal endotoxin release

Transition from acute to chronic inflammation is orchestrated by sustained production of cytokines (e.g., TNF-, IL-1, IL-6, CXCL8), continuous activation of macrophages, neutrophils, and T cells, defective resolution programmes, tissue remodelling driven by transforming growth factor-, and altered cell-death pathways

This methylation is crucial to regulate gene expression, neurotransmitter synthesis, myelin formation, and the methylation of histones and DNA (, , )

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