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Survodutide, a glucagon/GLP-1 dual agonist, met its primary endpoint of histological improvement in MASH at 48 weeks and also demonstrated a statistically significant improvement in fibrosis, a secondary endpoint

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This restoration was attributed to the normalization of both endothelium-dependent and nerve-mediated corpus cavernosum relaxation, along with reactivation of the cGMP signaling pathway and attenuation of cavernosal hypercontractility [48]