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Sarcosine is then oxidized to glycine in another FAD- and THF-dependent reaction catalyzed by sarcosine dehydrogenase (encoded by the SARDH gene)

In experiments performed under glucose minimal conditions with glutamate as the only nitrogen source, GDH2 deletion caused limited growth and impaired utilization of the specific amino acid leading to low amounts of intracellular ammonia [4]

Platelet hyperactivity and diabetes Platelet hyperreactivity is a crucial determinant in developing thrombotic diseases such as heart attack and ischemic stroke, which may be fatal consequences of diabetes

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Luckily, our bodies produce a secondary substance, glutathione, that helps minimize the toxic effects