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Emerging research has also highlighted the critical role of epigenetic and post-transcriptional regulation, particularly through microRNAs (miRNAs), which serve as key modulators of immune responses and microbial interactions in IBD, influencing not only immune modulation but also iron metabolism, oxidative stress, and epithelial integrity, all of which are critical to ferroptosis in IBD (94, 95)

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Additionally, animal studies have administered BPC-157 for periods exceeding 14 days without observing any significant safety concerns or adverse effects [16]

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