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In SURPASS-1, up to 51% of participants on the 15 mg dose achieved an A1C below 5.7% (the threshold for normal glucose metabolism)

10.1016/S0140-6736(23)01200-X

Preclinical investigations encompassing ethanol-induced gastric mucosal lesions, NSAID-mediated ulceration, cysteamine-induced duodenal injury, and inflammatory bowel disease analogues have reported associations between BPC157 exposure and reductions in mucosal lesion surface area, preservation of submucosal vascular architecture, and modulation of mucosal structural integrity

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