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Mitochondrial DNA programs lactylation of cGAS to induce IFN responses in patients with systemic lupus erythematosus

This plateau can be a signal to reassess treatment plans, including the possibility of weaning down the medication

This triple mechanism distinguishes it from existing agents such as semaglutide (a GLP-1 receptor agonist) or tirzepatide (a dual GIP/GLP-1 agonist), and is hypothesised to produce more pronounced metabolic effects, particularly in the liver

Brain Res 1187 , 116124, (2008)

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