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Ridker PM, Macfadyen JG, Glynn RJ et al (2018) Inhibition of interleukin-1 by canakinumab and cardiovascular outcomes in patients with chronic kidney disease

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The safety profile remained consistent whether patients continued or paused treatment, indicating that the drug does not accumulate or create cumulative organ damage over extended use

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We demonstrated that SYNCRIP deficiency sensitizes GBM cells to ferroptosis by inducing ROS accumulation, suggesting that ferroptosis is a key mechanism underlying the antitumor phenotype observed in SYNCRIP-deficient cells