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Its essential to consider these treatments' potential side effects and complications before proceeding

Kremer et al., 2024) with an indispensable role of STAT3 activation due to methylation at a CpG site in the GFAP promoter (Takizawa et al., 2001), and in TET1/2/3-mediated DNA demethylation for establishing neural stem cell identity and enabling glial (e.g., astrocyte and oligodendrocyte) differentiation (MacArthur et al., 2024)

the signal molecules (mTOR, PI3K/Akt, AMPK, Nrf-2/HO-1, HIF-1, Notch, RhoA and GSK-3)

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