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J., Reitsma, W

PGC-1 deregulation is linked with the development and progression of PD through increasing sensitivity of DNs to the effect of neurotoxins like MPTP

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Overall, our results suggest that the AP is responsible for the appetite-suppressing effects of GIPR agonism but that GIP receptors in the hypothalamus underlie the ability of GIPR antagonism to enhance the weight loss effects of GLP-1R and amylin receptor agonists

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