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In cells, GLP-1(936) activates the GLP-1R, which increases cAMP levels, subsequently activating PKA and CREB, leading to the promotion of insulin secretion

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It can potentially be a therapeutic agent for managing NDs, indicating a need for further research

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In the previous century, the underlined function of TMAO was the stabilization of proteins against various environmental stress factors, including high hydrostatic pressure [73]