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GRAIL normally mono-ubiquitinates CUL5 lysine K724 (65), which prevents K724 neddylation and results in CUL5 inhibition, but in autoimmune patients with diminished GRAIL expression, increased CUL5 activity leads to a more rapid pJAK1 degradation

Eyles, D

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Non-iron metalloporphyrins: potent antibacterial compounds that exploit haem/Hb uptake systems of pathogenic bacteria