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by proinflammatory factors and products of metabolic stress) and specifically to counter the declining beta cell population using therapies that reduce beta cell apoptosis or replenish through beta cell neogenesis

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GLP-1R agonists can improve left ventricular ejection fraction, myocardial contractility, coronary blood flow, cardiac output, and endothelial function while reducing infarction size and overall risks for a cardiovascular event