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Trillions of microbes living in the digestive tract influence everything from nutrient absorption to inflammation and hormone production

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Redox homeostasis genes modulate response to APR-246 in a p53-dependent and p53-independent manner Previous studies have characterized APR-246 as a modulator of redox homeostasis and inducer of ROS through depletion of key antioxidants, resulting in both p53-dependent and -independent cell death pathways such as ferroptosis [26,27,28,29]

There is extensive literature from the Diabetes Prevention Program and the Diabetes Prevention Program Outcomes Study that support intensive lifestyle interventions having positive effects on overall health outcomes [8, 9]