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With activation of normal PSCs, GLP-1R is markedly increased, as is multiple other incretin-related receptors

Over time, this energy shortfall sets off a cascade of downstream effects: synaptic dysfunction, impaired protein clearance, neuroinflammation, and ultimately the accumulation of amyloid- and tau pathology Importantly, imaging and metabolic studies consistently show that reductions in cerebral glucose metabolism and perfusion precede cognitive symptoms by years, often decades [8]

These medications work by mimicking the action of naturally occurring GLP-1, an incretin hormone released by the intestine in response to food intake

These benefits are updated and refined based on continuing feedback from team members across the system

Injectable GLP-1s are delivered directly into the bloodstream, bypassing the liver and resulting in a longer duration of action, while oral semaglutide is broken down more quickly and must be taken daily at a higher dose for comparable effects