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BPC157 exhibits broad tissue repair capacity, suggesting that its actions may not depend on cell type-specific regulation (e.g., myocytes or epithelial cells) but rather target universal injury responses

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Senescence was confirmed by SA--GAL assay(Dimri et al., 1995) and/or changes in morphology

Congenital hemolytic anemias attributed to defects in the biosynthesis of GSH (in the genes of GCL, GCS and glutathione reductase) highlight the key role of GSH in RBCs homeostasis [197,198]