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Mitohormesis encapsulates the idea that exposing mitochondria to a challenging (but not excessive) stressor leads to a recalibration of mitochondrial biology that subsequently protects the organelles against higher, normally harmful exposures to similar stressors in the future [164, 165]

Normally, these cytokines are used to trigger an inflammatory response against infection (such as a fever)

Selenocysteine confers the biochemical properties characteristic of the type I iodothyronine deiodinase

A.BlaM.Gligor PaneD.ZippenfeningS

However, this study did not directly validate whether ER or PR themselves serve as terminal targets for phosphorylation alterations induced by OS