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doi: 10.4161/oxim.3.4.12734 64 KaltsasAKoumenisAStavropoulosMKratirasZDeligiannisDAdamosKet al

This could enable the treatment of hypoxic tumors, which represent a major challenge in anticancer drug development

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Including enhancement of nucleotide excision repair capacity in melanocytes and keratinocytes following MT-1 treatment, reduction of UV-induced cyclobutane pyrimidine dimer formation, activation of antioxidant defence pathways, and upregulation of DNA repair enzyme expression downstream of MC1R-cAMP signalling

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