Blood-brain glucose transfer in Alzheimer's disease: effect of GLP-1 analog treatment
This is a great resource for both providers and patients navigating treatment with these powerful agents.
Users often share practical approaches, experimenting with dosing schedules to balance appetite control, side effects, and affordability
Coupled with the new dosage, the richness of the appetizer had me feeling nauseous even before the entree arrived
It has multiple actions including: potentiation of glucose-mediated insulin secretion mechanisms identified: increased -cell proliferation, resulting in an increase -cell mass (Fusco et al, 2017) stimulation of insulin biosynthesis at the translational level, helping to maintain -cell insulin stores and secretory capacity (Baggio & Drucker, 2007) because the GLP-1 effect is glucose-dependent (there is more insulin release when glucose levels are elevated, but less effect when glucose levels are normal), GLP-1 agonists have a lower risk for producing hypoglycemia compared to sulfonylureas (that chronically stimulate insulin release, independent of glucose concentration) suppression of postprandial glucagon release Evidence indicates that stimulation of pancreatic cells by GLP-1 increases their glucose sensitivity, resulting in less glucagon release at any glucose level (Baggio & Drucker, 2007)
