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In conclusion, the degradation of killed bacteria contributes more efficiently to improving cell viability under nutrient-deprived conditions than live bacteria, stimulating pathways such as glutathione (GSH) biosynthesis, compared to live bacteria [56]

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In HFD-fed KK-Ay mice, L-carnitine administration inhibited Srebp1c mRNA overexpression while increasing -hydroxybutyrate levels, suggesting concurrent lipogenesis suppression and -oxidation activation ( Hmgcr, Srebp2, Pcsk9, Ldlr ) in HFD-fed mice