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The best characterized of these paracrine intermediates are keratinocyte growth factor (KGF), which mediates GLP-2-induced colonic growth, and insulin-like growth factor-1 (IGF-1), which is essential for GLP-2s trophic effects in the small intestine (68)

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Specifically, it has been established that 50% to 70% of insulin secretion in response to oral glucose administration is due to the incretin effect [1]