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While increased ROS production can also be detrimental to the plant, at subtoxic doses, sulforaphane also activates the plants host defense mechanisms by inducing expression of stress response genes, such as heat shock proteins (Ferber et al., 2020)

One prominent example of these disorders is the cryopyrin-associated periodic syndrome (CAPS), which comprise a spectrum of disorders caused by gain-of-function mutations in the NLRP3 gene (also referred to cold-induced autoinflammatory syndrome 1, or CIAS1 gene) on chromosome 1q44, which encodes cryopyrin

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LTF can certainly modify iron homeostasis, critical for responsiveness to bacterial insult, and subsequent inflammation, as discussed previously (68)