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Uremic toxins and persistent ROS can activate NF-B, MAPK, and other stress-responsive pathways that enhance KEAP1 transcription ( in vivo (especially those that simulate IS accumulation) will be significant in confirming the therapeutic significance of AST and determining whether its protective effect on the kidney is applicable in different types of chronic kidney disease etiologies

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The final step in carnitine synthesis that occurs in liver and kidney depends on the enzyme -butyrobetaine hydroxylase, which may be deficient in children

Trends Microbiol 15 , 109118 (2007)