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PubMed , Biochemical Pharmacology , vol

GSH and glutaredoxins are concerned with detoxification of ROS, but they also transmit redox signals under drought stress (Meyer 2008)

Glutathione might inhibit the effects of cerebral infarction and enhance antiapoptotic signaling after ischemic stroke, suggesting that GSH may be a potent therapeutic antioxidant that can attenuate severe pathologies after ischemic stroke, and stimulating GSH synthesis through administration of GSH precursors and micronutrients like selenium can optimize GSH and GSH peroxidase for optimal antioxidant defense in cerebral ischemia (155, 156)

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