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These findings reveal that metabolic abnormalities may involve in the pathophysiological mechanism of preeclampsia, which suggests that in the future researches, more specific metabolic pathways need to be explored in preeclampsia based on animals and in vitro models, which is of great significance to the development of new metabolic drugs for the alleviation of preeclamptic symptoms
Group 1 ( n = 4) chow diet
However, when Cys 129 residue of PTPN22 is mutated, the disulfide bond cannot form and the enzyme is exposed by oxidation, resulting in inactivation (93)
