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Consistent with the previous results, IF results showed that CaOx-exo-stimulated macrophages were polarized into M1 phenotype, and METs formation was significantly increased, which could be reversed by using miR-93-3p inhibitor and si-NIK (Fig
ALCAR attenuated microglial activation and release of inflammatory mediators through balancing proinflammatory and anti-inflammatory cytokines, which subsequently enhanced the survival of mature neurons in the CA1, CA3, and PFC regions and improved cognitive functions in hemiparkinsonian rats
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