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First, the absence of denominator data precludes estimation of event incidence or risk ratios, as the total number of exposed individuals (to lithium, GLP-1 RAs, or both) is unknown

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Moreover the hormonal rearrangement intrinsic of pregnancy [17] redistributes the interrelationships of metabolic pathways that sustain fetus growth and differentiation, particularly in rats, that have a short pregnancy (about 17 d)