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To determine if the reduction in HGP in Dpp4 / mice was mediated through the actions of hepatocyte-derived DPP4, we then measured HGP in HFHC-fed, hepatocyte-specific Dpp4 -knockout mice ( Dpp4 hep/ )

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Therefore, while the fundamental mechanisms of LTP are preserved in epilepsy, their alteration through dysregulated calcium signaling leads to a paradoxical situation

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