PEER REVIEW The peer review history for this article is available at Supporting information ACKNOWLEDGMENTS The authors thank Paul Barlass, PhD, of Axis, a division of Spirit Medical Communications Group Ltd., and Sarah Stowell, a contract writer working on behalf of Axis who provided medical writing assistance under the direction of the authors, funded by Novo Nordisk A/S, Denmark, in accordance with Good Publication Practice 3 (GPP3) guidelines (www.ismpp.org/gpp3)
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Agonists, such as (Val8) GLP-1, showed a clear upregulation of LTP in area CA1 of the hippocampus, while the selective GLP-1R antagonist exendin (936) blocked LTP