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Regulation of transport pathways in tumor vessels: role of tumor type and microenvironment

N -methyl-D-aspartate (NMDA)-DR heteromers NMDA-D1R Heteromers Heteromers, consist of D1R and NMDAR, increase the phosphorylation of NR1 and NR2B subunits, surface insertion of NR2B, and NMDA-triggered Ca2+ upregulation via cAMP/PKA/RACK1-mediated Fyn activation (67, 68), and PKC/CAK/Src signaling (69), thus enhancing NMDAR-mediated currents

Activating GLP-1R inhibits PFKFB3-driven glycolysis and histone lactylation in TGF-1 + IL-1-stimulated fibroblasts Our results demonstrated that TGF-1 + IL-1 augmented PFKFB3-driven glycolysis, lactate production and histone lactylation in fibroblasts in vitro (Fig

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