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Similar to the adult rat TBI model study by Shuhmann et al., we observed significant decrease in concentrations of several neurometabolites in the ipsilateral hippocampus at 24 h post TBI

Drawn from a 150+ insight panel across 14 pathways

The mechanism of action of TREM2 agonists may involve the activation of downstream signaling pathways such as PI3K-AKT-mTOR, which regulate microglial metabolism and function to promote M2 polarization [302]

Consequently, it was found that these 12 compounds could act on 235 anti-AD targets, of which AKT and other targets were the core targets

The standard escalation in phase 2 trials started at either 2 mg or 4 mg weekly, with increases every four weeks for up to 12 weeks, reaching maximum doses of 8-12 mg