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They also stimulate the endothelial nitric oxide synthase (eNOS)/NO signaling pathway, decrease endothelial permeability, inhibit ACE activity, increase diuresis, regulate Ca 2+ levels in VSMCs or myocardial cells, inhibit the expression or activity of contractile and structural proteins, and open K + ATP -channels

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Although oxidative stress may not be the only cause of cell derangement and of disease, an ever-increasing number of disease conditions at several organs and systems, such as cancer, cardiovascular, neurodegenerative and endocrine diseases, are linked to this altered state