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We demonstrate that a FN-rich environment rewires MCs toward a dedifferentiated state, marked by reduced melanin production, slow-cycling, and increased motility, while COL I elicits features of a differentiated phenotype, promoting melanin synthesis but limiting migration

Cell growth, proliferation, and survival are governed by the HGF/c-MET signaling pathway, which operates through complex downstream signaling sequences

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The follicle then becomes a corpus luteum, which produces progesterone and estrogen