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This is due to the increased levels of cytokines that induce characteristic alterations in positive and negative APPs in the liver [44]
De-ethynylation of EE may, therefore, have contributed to the increased formation of 2-OH catechol estrogens and their methylation, although any contribution of this pathway would most likely have been small
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Elevated levels of CHAC1, often induced by ER stress, lead to reduced GSH levels, increased oxidative stress, and greater vulnerability to cell death processes like ferroptosis
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