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Transient stress or increased cytoplasmic TDP-43 triggers phase separation into toxic cytoplasmic droplets that block nuclear import, deplete nuclear TDP-43, and cause cell death, offering a present mechanistic model for ALS/FTD pathology (Gasset-Rosa et al., 2019)
Arch Phys Med Rehabil
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Semaglutide has already been approved as a CVD risk-reducer, and these results suggest that tirzepatide is on a similar path for HFpEF
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