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In OA models, ACSL4 inhibition dramatically decreases ferroptosis-associated lipid peroxidation ( in vitro models, inhibiting cartilage chondroptosis and improving OA ( Excessive lipid metabolic reprogramming in OA significantly raises ferroptosis risk, so targeting lipid synthesis, transport, and degradation represents a promising upstream approach to reducing ferroptotic damage

Considering the present findings and those of previous studies, chronic antioxidant reduction may contribute to the pathogenesis of late radiation effects

38 (3), e3502

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Researchers have found that exercise clears toxins that are produced in our bodies and reduces inflammation