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Upon the release of zonulin into the lumen as a result of intestinal exposure to gliadin, zonulin can interact with two receptors, epidermal growth factor receptor (EGFR) and protease-activated receptor 2 (PAR2), which results in increased intestinal permeability (27, 32)

Addressing this accumulated damage requires regenerative processes the peptide appears to stimulate

10.1038/s41598-020-74022-y 29 HsiehM

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These data are consistent with studies in human dermal papilla cells, which reported that the phosphorylation of ERK1/2 was changed in terms of intensity and the phosphorylation of P38, JNK and AKT was fixed in dermal papilla cells treated with VEGF or VEGFr-2 neutralizing antibodies [34]