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Abrogation of BRAF V600E -induced senescence by PI3K pathway activation contributes to melanomagenesis

Cellular and molecular analyses were beyond the scope of this study

Epidemiologic research can identify associations, yet it does not always prove causation

Kidney stones, insulin resistance, and metabolic syndrome

In both pathways, cysteine-dependent proteases known as caspases execute apoptotic cell death by cleaving key structural and regulatory proteins, primarily through the action of caspase-3 (CASP3) and caspase-7 (CASP7), with caspase-6 (CASP6) contributing to downstream substrate cleavage and apoptotic progression