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These genetic predispositions may inform whether you are likely to experience robust glucose response, modest response, or potential side effects to GLP-1 therapy, though genetic testing does not predict exact medication outcomes

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Their mechanisms of action include modulation of aryl hydrocarbon receptor (AHR) signaling, impact on estrogen receptor (ER)-dependent signaling in breast cancer cells, antagonism of the androgen receptor in prostate cancer, epigenetic regulation of gene expression and modulation of DNA methylation and histone modifications [156]