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See our GHK-Cu copper peptide guide for background

The inhibitory effect of glucocorticoids on corticotroph POMC expression appears, in the rat POMC promoter, to be dependent on a glucocorticoid response element partially overlapping the CCAAT box (27)

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Furthermore, AKT potentiates the activity of hexokinase and phosphofructokinase enzymes, which catalyse rate-limiting steps of glycolysis, in order to induce glucose consumption to branching pathways [7-9]