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While emerging evidence establishes ferroptosis as a pivotal mediator of SLE nephritis (LN) pathogenesislinking iron dyshomeostasis, immune cell dysfunction, and renal injuryand identifies GPX4 as a key regulatory checkpoint, several critical, insufficiently addressed research gaps persist

Research published on PubMed documents GIP receptor involvement in bone metabolism, gastric motility, and neuroprotective signaling beyond metabolic effects

UK Biobank data are available through a procedure described at Registered researchers whose institutions have Data Use and Registration Agreements in place with All of Us that include the Controlled Tier can access genomic data

Your Partner in Health Starting a new medication is a journey, and at GobyMeds, were here for you every step of the way

Understanding the role of Gpx4 in immune cells can provide a better understanding of potential factors underlying immune cell ferroptosis [80]